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肿瘤微环境中调节性T细胞通过分泌白细胞介素-10和转化生长因子-β,抑制效应T细胞的杀伤功能,从而介导免疫逃逸。靶向PD-1/PD-L1信号通路的免疫检查点抑制剂已在多种实体瘤中展现出持久的客观缓解率,但其疗效与肿瘤突变负荷及微卫星不稳定性状态密切相关。
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Regulatory T cells in the tumor microenvironment suppress the cytotoxic function of effector T cells by secreting interleukin-10 and transforming growth factor-β, thereby mediating immune evasion. Immune checkpoint inhibitors targeting the PD-1/PD-L1 signaling pathway have demonstrated durable objective response rates in various solid tumors, but their efficacy is closely associated with tumor mutational burden and microsatellite instability status.

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快速模式Regulatory T cells in the tumor microenvironment suppress the cytotoxic function of effector T cells by secreting interleukin-10 and transforming growth factor-β, thereby mediating immune evasion.

深度模式Within the tumor microenvironment, regulatory T cells suppress effector T cell cytotoxicity through secretion of interleukin-10 and transforming growth factor-β, thereby mediating immune evasion.

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